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Table representation of search results timeline featuring number of search results per year.
Year | Number of Results |
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2014 | 1 |
2015 | 3 |
2016 | 2 |
2024 | 0 |
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Hypoxia Increases IGFBP-1 Phosphorylation Mediated by mTOR Inhibition.
Mol Endocrinol. 2016 Feb;30(2):201-16. doi: 10.1210/me.2015-1194. Epub 2015 Dec 29.
Mol Endocrinol. 2016.
PMID: 26714229
Free PMC article.
IGFBP-1 hyperphosphorylation in response to leucine deprivation is mediated by the AAR pathway.
Malkani N, Jansson T, Gupta MB.
Malkani N, et al.
Mol Cell Endocrinol. 2015 Sep 5;412:182-95. doi: 10.1016/j.mce.2015.04.031. Epub 2015 May 5.
Mol Cell Endocrinol. 2015.
PMID: 25957086
Free PMC article.
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Increased IGFBP-1 phosphorylation in response to leucine deprivation is mediated by CK2 and PKC.
Malkani N, Biggar K, Shehab MA, Li SS, Jansson T, Gupta MB.
Malkani N, et al.
Mol Cell Endocrinol. 2016 Apr 15;425:48-60. doi: 10.1016/j.mce.2015.12.006. Epub 2015 Dec 28.
Mol Cell Endocrinol. 2016.
PMID: 26733150
Free PMC article.
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Liver mTOR controls IGF-I bioavailability by regulation of protein kinase CK2 and IGFBP-1 phosphorylation in fetal growth restriction.
Abu Shehab M, Damerill I, Shen T, Rosario FJ, Nijland M, Nathanielsz PW, Kamat A, Jansson T, Gupta MB.
Abu Shehab M, et al.
Endocrinology. 2014 Apr;155(4):1327-39. doi: 10.1210/en.2013-1759. Epub 2014 Jan 17.
Endocrinology. 2014.
PMID: 24437487
Free PMC article.
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