Bruschi SA - Search Results

1

Cysteine S-conjugate β-lyases: important roles in the metabolism of naturally occurring sulfur and selenium-containing compounds, xenobiotics and anticancer agents.

Cooper AJ, Krasnikov BF, Niatsetskaya ZV, Pinto JT, Callery PS, Villar MT, Artigues A, Bruschi SA. Cooper AJ, et al. Among authors: bruschi sa. Amino Acids. 2011 Jun;41(1):7-27. doi: 10.1007/s00726-010-0552-0. Epub 2010 Mar 22. Amino Acids. 2011. PMID: 20306345 Free PMC article. Review.

2

Measurement of cysteine S-conjugate β-lyase activity.

Cooper AJ, Krasnikov BF, Pinto JT, Bruschi SA. Cooper AJ, et al. Among authors: bruschi sa. Curr Protoc Toxicol. 2010 May;Chapter 4:Unit 4.36. doi: 10.1002/0471140856.tx0436s44. Curr Protoc Toxicol. 2010. PMID: 20949433 Free PMC article.

3

Mitochondrial aspartate aminotransferase catalyses cysteine S-conjugate beta-lyase reactions.

Cooper AJ, Bruschi SA, Iriarte A, Martinez-Carrion M. Cooper AJ, et al. Among authors: bruschi sa. Biochem J. 2002 Nov 15;368(Pt 1):253-61. doi: 10.1042/BJ20020531. Biochem J. 2002. PMID: 12137566 Free PMC article.

4

Toxic, halogenated cysteine S-conjugates and targeting of mitochondrial enzymes of energy metabolism.

Cooper AJ, Bruschi SA, Anders MW. Cooper AJ, et al. Among authors: bruschi sa. Biochem Pharmacol. 2002 Aug 15;64(4):553-64. doi: 10.1016/s0006-2952(02)01076-6. Biochem Pharmacol. 2002. PMID: 12167474 Review.

5

Human mitochondrial and cytosolic branched-chain aminotransferases are cysteine S-conjugate beta-lyases, but turnover leads to inactivation.

Cooper AJ, Bruschi SA, Conway M, Hutson SM. Cooper AJ, et al. Among authors: bruschi sa. Biochem Pharmacol. 2003 Jan 15;65(2):181-92. doi: 10.1016/s0006-2952(02)01513-7. Biochem Pharmacol. 2003. PMID: 12504794

6

Co-purification of mitochondrial HSP70 and mature protein disulfide isomerase with a functional rat kidney high-M(r) cysteine S-conjugate beta-lyase.

Cooper AJ, Wang J, Gartner CA, Bruschi SA. Cooper AJ, et al. Among authors: bruschi sa. Biochem Pharmacol. 2001 Nov 15;62(10):1345-53. doi: 10.1016/s0006-2952(01)00802-4. Biochem Pharmacol. 2001. PMID: 11709194

7

BCL-xL overexpression effectively protects against tetrafluoroethylcysteine-induced intramitochondrial damage and cell death.

Ho HK, Hu ZH, Tzung SP, Hockenbery DM, Fausto N, Nelson SD, Bruschi SA. Ho HK, et al. Among authors: bruschi sa. Biochem Pharmacol. 2005 Jan 1;69(1):147-57. doi: 10.1016/j.bcp.2004.08.030. Biochem Pharmacol. 2005. PMID: 15588723

8

Cell culture model for acetaminophen-induced hepatocyte death in vivo.

Pierce RH, Franklin CC, Campbell JS, Tonge RP, Chen W, Fausto N, Nelson SD, Bruschi SA. Pierce RH, et al. Among authors: bruschi sa. Biochem Pharmacol. 2002 Aug 1;64(3):413-24. doi: 10.1016/s0006-2952(02)01180-2. Biochem Pharmacol. 2002. PMID: 12147292

9

Mitochondrial HSP60 (P1 protein) and a HSP70-like protein (mortalin) are major targets for modification during S-(1,1,2,2-tetrafluoroethyl)-L-cysteine-induced nephrotoxicity.

Bruschi SA, West KA, Crabb JW, Gupta RS, Stevens JL. Bruschi SA, et al. J Biol Chem. 1993 Nov 5;268(31):23157-61. J Biol Chem. 1993. PMID: 7901206 Free article.

10

Cytosolic heat shock proteins and heme oxygenase-1 are preferentially induced in response to specific and localized intramitochondrial damage by tetrafluoroethylcysteine.

Ho HK, Jia Y, Coe KJ, Gao Q, Doneanu CE, Hu Z, Bammler TK, Beyer RP, Fausto N, Bruschi SA, Nelson SD. Ho HK, et al. Among authors: bruschi sa. Biochem Pharmacol. 2006 Jun 28;72(1):80-90. doi: 10.1016/j.bcp.2006.03.019. Epub 2006 Mar 29. Biochem Pharmacol. 2006. PMID: 16678137

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