Christiansen DH - Search Results

1

Genetic pathways in therapy-related myelodysplasia and acute myeloid leukemia.

Pedersen-Bjergaard J, Andersen MK, Christiansen DH, Nerlov C. Pedersen-Bjergaard J, et al. Among authors: christiansen dh. Blood. 2002 Mar 15;99(6):1909-12. doi: 10.1182/blood.v99.6.1909. Blood. 2002. PMID: 11877259 Free article.

2

Mutations of AML1 are common in therapy-related myelodysplasia following therapy with alkylating agents and are significantly associated with deletion or loss of chromosome arm 7q and with subsequent leukemic transformation.

Christiansen DH, Andersen MK, Pedersen-Bjergaard J. Christiansen DH, et al. Blood. 2004 Sep 1;104(5):1474-81. doi: 10.1182/blood-2004-02-0754. Epub 2004 May 13. Blood. 2004. PMID: 15142876 Free article.

3

Centromeric breakage and highly rearranged chromosome derivatives associated with mutations of TP53 are common in therapy-related MDS and AML after therapy with alkylating agents: an M-FISH study.

Andersen MK, Christiansen DH, Pedersen-Bjergaard J. Andersen MK, et al. Among authors: christiansen dh. Genes Chromosomes Cancer. 2005 Apr;42(4):358-71. doi: 10.1002/gcc.20145. Genes Chromosomes Cancer. 2005. PMID: 15645489

4

Mutations of the PTPN11 gene in therapy-related MDS and AML with rare balanced chromosome translocations.

Christiansen DH, Desta F, Andersen MK, Pedersen-Bjergaard J. Christiansen DH, et al. Genes Chromosomes Cancer. 2007 Jun;46(6):517-21. doi: 10.1002/gcc.20426. Genes Chromosomes Cancer. 2007. PMID: 17330262

5

Pedersen-Bjergaard J, Andersen MK, Christiansen DH. Pedersen-Bjergaard J, et al. Among authors: christiansen dh. Blood. 2000 Jun 1;95(11):3273-9. Blood. 2000. PMID: 10828005 Free article. Review.

6

Mutations with loss of heterozygosity of p53 are common in therapy-related myelodysplasia and acute myeloid leukemia after exposure to alkylating agents and significantly associated with deletion or loss of 5q, a complex karyotype, and a poor prognosis.

Christiansen DH, Andersen MK, Pedersen-Bjergaard J. Christiansen DH, et al. J Clin Oncol. 2001 Mar 1;19(5):1405-13. doi: 10.1200/JCO.2001.19.5.1405. J Clin Oncol. 2001. PMID: 11230485

7

Duplication or amplification of chromosome band 11q23, including the unrearranged MLL gene, is a recurrent abnormality in therapy-related MDS and AML, and is closely related to mutation of the TP53 gene and to previous therapy with alkylating agents.

Andersen MK, Christiansen DH, Kirchhoff M, Pedersen-Bjergaard J. Andersen MK, et al. Among authors: christiansen dh. Genes Chromosomes Cancer. 2001 May;31(1):33-41. doi: 10.1002/gcc.1115. Genes Chromosomes Cancer. 2001. PMID: 11284033

8

Andersen MK, Christiansen DH, Jensen BA, Ernst P, Hauge G, Pedersen-Bjergaard J. Andersen MK, et al. Among authors: christiansen dh. Br J Haematol. 2001 Sep;114(3):539-43. doi: 10.1046/j.1365-2141.2001.03000.x. Br J Haematol. 2001. PMID: 11552977 Free article. Review.

9

Internal tandem duplications of the FLT3 and MLL genes are mainly observed in atypical cases of therapy-related acute myeloid leukemia with a normal karyotype and are unrelated to type of previous therapy.

Christiansen DH, Pedersen-Bjergaard J. Christiansen DH, et al. Leukemia. 2001 Dec;15(12):1848-51. doi: 10.1038/sj.leu.2402246. Leukemia. 2001. PMID: 11753604

10

Causality of myelodysplasia and acute myeloid leukemia and their genetic abnormalities.

Pedersen-Bjergaard J, Christiansen DH, Andersen MK, Skovby F. Pedersen-Bjergaard J, et al. Among authors: christiansen dh. Leukemia. 2002 Nov;16(11):2177-84. doi: 10.1038/sj.leu.2402764. Leukemia. 2002. PMID: 12399959 Review.

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