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20-hydroxyeicosatetraenoic acid causes endothelial dysfunction via eNOS uncoupling.
Cheng J, Ou JS, Singh H, Falck JR, Narsimhaswamy D, Pritchard KA Jr, Schwartzman ML. Cheng J, et al. Among authors: falck jr. Am J Physiol Heart Circ Physiol. 2008 Feb;294(2):H1018-26. doi: 10.1152/ajpheart.01172.2007. Epub 2007 Dec 21. Am J Physiol Heart Circ Physiol. 2008. PMID: 18156192 Free article.
Induction of angiotensin-converting enzyme and activation of the renin-angiotensin system contribute to 20-hydroxyeicosatetraenoic acid-mediated endothelial dysfunction.
Cheng J, Garcia V, Ding Y, Wu CC, Thakar K, Falck JR, Ramu E, Schwartzman ML. Cheng J, et al. Among authors: falck jr. Arterioscler Thromb Vasc Biol. 2012 Aug;32(8):1917-24. doi: 10.1161/ATVBAHA.112.248344. Epub 2012 Jun 21. Arterioscler Thromb Vasc Biol. 2012. PMID: 22723444 Free PMC article.
Elevated 20-HETE impairs coronary collateral growth in metabolic syndrome via endothelial dysfunction.
Joseph G, Soler A, Hutcheson R, Hunter I, Bradford C, Hutcheson B, Gotlinger KH, Jiang H, Falck JR, Proctor S, Schwartzman ML, Rocic P. Joseph G, et al. Among authors: falck jr. Am J Physiol Heart Circ Physiol. 2017 Mar 1;312(3):H528-H540. doi: 10.1152/ajpheart.00561.2016. Epub 2016 Dec 23. Am J Physiol Heart Circ Physiol. 2017. PMID: 28011587 Free PMC article.
683 results