Wang X - Search Results

1

MicroRNA-32 upregulation by 1,25-dihydroxyvitamin D3 in human myeloid leukemia cells leads to Bim targeting and inhibition of AraC-induced apoptosis.

Gocek E, Wang X, Liu X, Liu CG, Studzinski GP. Gocek E, et al. Among authors: wang x. Cancer Res. 2011 Oct 1;71(19):6230-9. doi: 10.1158/0008-5472.CAN-11-1717. Epub 2011 Aug 4. Cancer Res. 2011. PMID: 21816906 Free PMC article.

2

Signaling of monocytic differentiation by a non-hypercalcemic analog of vitamin D3, 1,25(OH)2-5,6 trans-16-ene-vitamin D3, involves nuclear vitamin D receptor (nVDR) and non-nVDR-mediated pathways.

Ji Y, Wang X, Donnelly RJ, Uskokovic MR, Studzinski GP. Ji Y, et al. Among authors: wang x. J Cell Physiol. 2002 May;191(2):198-207. doi: 10.1002/jcp.10091. J Cell Physiol. 2002. PMID: 12064463

3

Jun N-terminal kinase pathway enhances signaling of monocytic differentiation of human leukemia cells induced by 1,25-dihydroxyvitamin D3.

Wang Q, Wang X, Studzinski GP. Wang Q, et al. Among authors: wang x. J Cell Biochem. 2003 Aug 15;89(6):1087-101. doi: 10.1002/jcb.10595. J Cell Biochem. 2003. PMID: 12898508

4

Kinase suppressor of RAS (KSR) amplifies the differentiation signal provided by low concentrations 1,25-dihydroxyvitamin D3.

Wang X, Studzinski GP. Wang X, et al. J Cell Physiol. 2004 Mar;198(3):333-42. doi: 10.1002/jcp.10443. J Cell Physiol. 2004. PMID: 14755538

5

Up-regulation of Egr1 by 1,25-dihydroxyvitamin D3 contributes to increased expression of p35 activator of cyclin-dependent kinase 5 and consequent onset of the terminal phase of HL60 cell differentiation.

Chen F, Wang Q, Wang X, Studzinski GP. Chen F, et al. Among authors: wang x, wang q. Cancer Res. 2004 Aug 1;64(15):5425-33. doi: 10.1158/0008-5472.CAN-04-0806. Cancer Res. 2004. PMID: 15289351

This laboratory has also provided evidence that the extracellular signal-regulated kinase/mitogen-activated protein kinase pathway is active in the early (24-48 h) stages of HL60 cell differentiation induced by 1,25D(3) but declines in the later, terminal phase of this form of di …

This laboratory has also provided evidence that the extracellular signal-regulated kinase/mitogen-activated protein kinase pathway is active …

6

The rationale for deltanoids in therapy for myeloid leukemia: role of KSR-MAPK-C/EBP pathway.

Studzinski GP, Wang X, Ji Y, Wang Q, Zhang Y, Kutner A, Harrison JS. Studzinski GP, et al. Among authors: wang x, wang q. J Steroid Biochem Mol Biol. 2005 Oct;97(1-2):47-55. doi: 10.1016/j.jsbmb.2005.06.010. Epub 2005 Jul 25. J Steroid Biochem Mol Biol. 2005. PMID: 16046262 Free PMC article.

7

The requirement for and changing composition of the activating protein-1 transcription factor during differentiation of human leukemia HL60 cells induced by 1,25-dihydroxyvitamin D3.

Wang X, Studzinski GP. Wang X, et al. Cancer Res. 2006 Apr 15;66(8):4402-9. doi: 10.1158/0008-5472.CAN-05-3109. Cancer Res. 2006. PMID: 16618766 Free PMC article.

8

Raf-1 signaling is required for the later stages of 1,25-dihydroxyvitamin D3-induced differentiation of HL60 cells but is not mediated by the MEK/ERK module.

Wang X, Studzinski GP. Wang X, et al. J Cell Physiol. 2006 Nov;209(2):253-60. doi: 10.1002/jcp.20731. J Cell Physiol. 2006. PMID: 16883571 Free PMC article.

9

Resistance to 1,25D-induced differentiation in human acute myeloid leukemia HL60-40AF cells is associated with reduced transcriptional activity and nuclear localization of the vitamin D receptor.

Garay E, Donnelly R, Wang X, Studzinski GP. Garay E, et al. Among authors: wang x. J Cell Physiol. 2007 Dec;213(3):816-25. doi: 10.1002/jcp.21150. J Cell Physiol. 2007. PMID: 17520689 Free PMC article.

10

Expression of human kinase suppressor of Ras 2 (hKSR-2) gene in HL60 leukemia cells is directly upregulated by 1,25-dihydroxyvitamin D(3) and is required for optimal cell differentiation.

Wang X, Wang TT, White JH, Studzinski GP. Wang X, et al. Among authors: wang tt. Exp Cell Res. 2007 Aug 15;313(14):3034-45. doi: 10.1016/j.yexcr.2007.05.021. Epub 2007 May 31. Exp Cell Res. 2007. PMID: 17599832 Free PMC article.

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