Wu-Baer F - Search Results

1

The BRCA1/BARD1 heterodimer assembles polyubiquitin chains through an unconventional linkage involving lysine residue K6 of ubiquitin.

Wu-Baer F, Lagrazon K, Yuan W, Baer R. Wu-Baer F, et al. J Biol Chem. 2003 Sep 12;278(37):34743-6. doi: 10.1074/jbc.C300249200. Epub 2003 Jul 30. J Biol Chem. 2003. PMID: 12890688 Free article.

2

BRCA1/BARD1 inhibition of mRNA 3' processing involves targeted degradation of RNA polymerase II.

Kleiman FE, Wu-Baer F, Fonseca D, Kaneko S, Baer R, Manley JL. Kleiman FE, et al. Among authors: wu baer f. Genes Dev. 2005 May 15;19(10):1227-37. doi: 10.1101/gad.1309505. Genes Dev. 2005. PMID: 15905410 Free PMC article.

3

The UBXN1 protein associates with autoubiquitinated forms of the BRCA1 tumor suppressor and inhibits its enzymatic function.

Wu-Baer F, Ludwig T, Baer R. Wu-Baer F, et al. Mol Cell Biol. 2010 Jun;30(11):2787-98. doi: 10.1128/MCB.01056-09. Epub 2010 Mar 29. Mol Cell Biol. 2010. PMID: 20351172 Free PMC article.

4

Effect of DNA damage on a BRCA1 complex.

Wu-Baer F, Baer R. Wu-Baer F, et al. Nature. 2001 Nov 1;414(6859):36. doi: 10.1038/35102118. Nature. 2001. PMID: 11689934

5

Mono- versus polyubiquitination: differential control of p53 fate by Mdm2.

Li M, Brooks CL, Wu-Baer F, Chen D, Baer R, Gu W. Li M, et al. Among authors: wu baer f. Science. 2003 Dec 12;302(5652):1972-5. doi: 10.1126/science.1091362. Science. 2003. PMID: 14671306

6

The BRCT Domains of the BRCA1 and BARD1 Tumor Suppressors Differentially Regulate Homology-Directed Repair and Stalled Fork Protection.

Billing D, Horiguchi M, Wu-Baer F, Taglialatela A, Leuzzi G, Nanez SA, Jiang W, Zha S, Szabolcs M, Lin CS, Ciccia A, Baer R. Billing D, et al. Among authors: wu baer f. Mol Cell. 2018 Oct 4;72(1):127-139.e8. doi: 10.1016/j.molcel.2018.08.016. Epub 2018 Sep 20. Mol Cell. 2018. PMID: 30244837 Free PMC article.

7

Activation of DSB processing requires phosphorylation of CtIP by ATR.

Peterson SE, Li Y, Wu-Baer F, Chait BT, Baer R, Yan H, Gottesman ME, Gautier J. Peterson SE, et al. Among authors: wu baer f. Mol Cell. 2013 Feb 21;49(4):657-67. doi: 10.1016/j.molcel.2012.11.020. Epub 2012 Dec 27. Mol Cell. 2013. PMID: 23273981 Free PMC article.

8

REV1-Polζ maintains the viability of homologous recombination-deficient cancer cells through mutagenic repair of PRIMPOL-dependent ssDNA gaps.

Taglialatela A, Leuzzi G, Sannino V, Cuella-Martin R, Huang JW, Wu-Baer F, Baer R, Costanzo V, Ciccia A. Taglialatela A, et al. Among authors: wu baer f. Mol Cell. 2021 Oct 7;81(19):4008-4025.e7. doi: 10.1016/j.molcel.2021.08.016. Epub 2021 Sep 10. Mol Cell. 2021. PMID: 34508659 Free PMC article.

9

CtIP is essential for early B cell proliferation and development in mice.

Liu X, Wang XS, Lee BJ, Wu-Baer FK, Lin X, Shao Z, Estes VM, Gautier J, Baer R, Zha S. Liu X, et al. Among authors: wu baer fk. J Exp Med. 2019 Jul 1;216(7):1648-1663. doi: 10.1084/jem.20181139. Epub 2019 May 16. J Exp Med. 2019. PMID: 31097467 Free PMC article.

10

CtIP-mediated DNA resection is dispensable for IgH class switch recombination by alternative end-joining.

Wang XS, Zhao J, Wu-Baer F, Shao Z, Lee BJ, Cupo OM, Rabadan R, Gautier J, Baer R, Zha S. Wang XS, et al. Among authors: wu baer f. Proc Natl Acad Sci U S A. 2020 Oct 13;117(41):25700-25711. doi: 10.1073/pnas.2010972117. Epub 2020 Sep 28. Proc Natl Acad Sci U S A. 2020. PMID: 32989150 Free PMC article.

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