Abstract
Plaques that form in the brains of Alzheimer patients are made of deposits of the amyloid-beta peptide. We analyze the time evolution of amyloid-beta deposition in immunostained brain slices from transgenic mice. We find that amyloid-beta deposits appear in clusters whose characteristic size increases from 14 microm in 8-month-old mice to 22 microm in 12-month-old mice. We show that the clustering has implications for the biological growth of amyloid-beta by presenting a growth model that accounts for the experimentally observed structure of individual deposits and predicts the formation of clusters of deposits and their time evolution.
Publication types
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Research Support, Non-U.S. Gov't
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Research Support, U.S. Gov't, Non-P.H.S.
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Research Support, U.S. Gov't, P.H.S.
MeSH terms
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Alzheimer Disease / etiology*
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Alzheimer Disease / metabolism*
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Alzheimer Disease / pathology
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Amyloid beta-Peptides / chemistry
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Amyloid beta-Peptides / genetics
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Amyloid beta-Peptides / metabolism*
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Animals
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Biophysical Phenomena
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Biophysics
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Disease Models, Animal
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Humans
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Immunohistochemistry
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Kinetics
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Macromolecular Substances
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Mice
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Mice, Transgenic
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Particle Size
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Plaque, Amyloid / chemistry
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Plaque, Amyloid / metabolism*
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Plaque, Amyloid / pathology*
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Time Factors
Substances
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Amyloid beta-Peptides
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Macromolecular Substances