Human intestinal mast cells produce IL-5 in vitro upon IgE receptor cross-linking and in vivo in the course of intestinal inflammatory disease

Eur J Immunol. 1999 May;29(5):1496-503. doi: 10.1002/(SICI)1521-4141(199905)29:05<1496::AID-IMMU1496>3.0.CO;2-5.

Abstract

IL-5, known to be produced by T lymphocytes and eosinophils, is a key regulator of intestinal diseases such as parasitosis or eosinophilic gastroenteritis. Here we examined if mast cells contribute to the IL-5 production in human intestinal mucosa. The number of IL-5-positive lamina propria cells was substantially higher in patients with intestinal inflammatory diseases (5.3 +/- 4.6%, n = 17) compared to healthy controls (0.5 +/- 0.9%, n = 8, p < 0.01). In patients, the IL-5-positive cells were eosinophils (70 +/- 13%) and mast cells (29 +/- 14%), whereas in controls all IL-5-positive cells were eosinophils. IL-5-positive T cells were not detected, likely because they do not store IL-5. In vitro studies with isolated human intestinal mast cells and eosinophils showed that mast cells do not produce IL-5 constitutively, but release high amounts of IL-5 (315 +/- 115 pg/10(6) cells) following IgE receptor cross-linking, compared to activated eosinophils (24 +/- 5 pg/10(6) cells). Inhibitor studies suggest a regulation of IL-5 production at the transcriptional level. In conclusion our data demonstrate that activated mast cells are a potent source of IL-5 in the human intestinal mucosa.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Adult
  • Aged
  • Cells, Cultured
  • Colitis, Ulcerative / metabolism*
  • Colitis, Ulcerative / pathology
  • Crohn Disease / metabolism*
  • Crohn Disease / pathology
  • Cross-Linking Reagents
  • Eosinophils / metabolism
  • Female
  • Food Hypersensitivity / metabolism*
  • Food Hypersensitivity / pathology
  • Humans
  • Interleukin-5 / biosynthesis*
  • Interleukin-5 / genetics
  • Intestinal Mucosa / cytology
  • Male
  • Mast Cells / cytology
  • Mast Cells / metabolism*
  • Middle Aged
  • RNA, Messenger
  • Receptors, IgE / metabolism*

Substances

  • Cross-Linking Reagents
  • Interleukin-5
  • RNA, Messenger
  • Receptors, IgE