Cartilage-hair hypoplasia syndrome: increased apoptosis of T lymphocytes is associated with altered expression of Fas (CD95), FasL (CD95L), IAP, Bax, and Bcl2

J Clin Immunol. 1999 Nov;19(6):428-34. doi: 10.1023/a:1020563018864.

Abstract

Cartilage-hair hypoplasia (CHH) is a rare autosomal recessive short-limbed dwarfism associated with thin and sparse hair and cell mediated or combined immunodeficiency. However, the basis of immune deficiency in CHH is unclear. In this study, we investigated a role of apoptosis in immunodeficiency in a patient with CHH. An increased apoptosis of both CD4+ and CD8+ T cells, as determined by TUNEL assay, was observed in CHH compared to an age-matched healthy dwarf control. Increased apoptosis in CHH was associated with increased expression of Fas (CD95), CD95L, and Bax and decreased expression of Bcl-2 and inhibitor of apoptosis protein (IAP) compared to the control. These data suggest that lymphopenia and immunodeficiency in CHH may be, at least in part, due to increased apoptosis of T cells, possibly through the Fas/ FasL signaling pathway.

Publication types

  • Case Reports

MeSH terms

  • Apoptosis*
  • Dwarfism / immunology*
  • Fas Ligand Protein
  • Hair Diseases / immunology*
  • Humans
  • Immunologic Deficiency Syndromes / immunology*
  • Male
  • Membrane Glycoproteins / analysis*
  • Middle Aged
  • Proteins / analysis*
  • Proto-Oncogene Proteins / analysis*
  • Proto-Oncogene Proteins c-bcl-2 / analysis*
  • T-Lymphocytes / physiology*
  • X-Linked Inhibitor of Apoptosis Protein
  • bcl-2-Associated X Protein
  • fas Receptor / analysis*

Substances

  • BAX protein, human
  • FASLG protein, human
  • Fas Ligand Protein
  • Membrane Glycoproteins
  • Proteins
  • Proto-Oncogene Proteins
  • Proto-Oncogene Proteins c-bcl-2
  • X-Linked Inhibitor of Apoptosis Protein
  • XIAP protein, human
  • bcl-2-Associated X Protein
  • fas Receptor