The effect of acute immobilization stress on the abundance of corticotropin-releasing factor receptor in lymphoid organs

J Neuroimmunol. 2000 Mar 1;103(2):153-64. doi: 10.1016/s0165-5728(99)00229-5.

Abstract

We have previously found a dramatic increase of corticotropin-releasing factor receptor (CRF-R1) production in splenic neutrophils of male C57BL/6N mice after application of an immunological stimulus. We demonstrate here that immobilization, a predominantly psychological stress, exhibited a similar effect. Shortly after 90 min of immobilization, the number of splenic CRF-RI+ cells was transiently increased by nearly 8-fold, while it was reduced in thymus and unchanged in lymph nodes. The CRF-R1+ cells were detected by an affinity-purified polyclonal antibody directed against the N-terminus of CRF-R1, and identified as neutrophils, eosinophils or their immature precursors on the basis of their nuclear shapes, Wright-Giemsa staining and colocalization of CRF-R1 with the ER-MP58 antigen.

MeSH terms

  • Animals
  • Cell Count
  • Eosinophils / cytology
  • Eosinophils / metabolism
  • Granulocytes / cytology
  • Granulocytes / metabolism
  • Hematopoietic Stem Cells / cytology
  • Hematopoietic Stem Cells / metabolism
  • Immobilization / physiology
  • Lymph Nodes / cytology
  • Lymph Nodes / metabolism*
  • Male
  • Mice
  • Mice, Inbred C57BL
  • Neutrophils / cytology
  • Neutrophils / metabolism
  • Receptors, Corticotropin-Releasing Hormone / metabolism*
  • Spleen / cytology
  • Spleen / metabolism*
  • Stress, Physiological / immunology
  • Stress, Physiological / metabolism*
  • Thymus Gland / cytology
  • Thymus Gland / metabolism*

Substances

  • Receptors, Corticotropin-Releasing Hormone
  • CRF receptor type 1