beta-adrenergic receptor signaling: an acute compensatory adjustment-inappropriate for the chronic stress of heart failure? Insights from Gsalpha overexpression and other genetically engineered animal models

Circ Res. 2000 Mar 17;86(5):502-6. doi: 10.1161/01.res.86.5.502.
No abstract available

Publication types

  • Research Support, U.S. Gov't, P.H.S.
  • Review

MeSH terms

  • Animals
  • Chronic Disease
  • Disease Models, Animal
  • GTP-Binding Protein alpha Subunits, Gs / genetics*
  • Gene Expression
  • Genetic Engineering
  • Heart Failure / genetics
  • Heart Failure / physiopathology*
  • Receptors, Adrenergic, beta / physiology*
  • Signal Transduction / physiology*
  • Stress, Physiological / genetics
  • Stress, Physiological / physiopathology*

Substances

  • Receptors, Adrenergic, beta
  • GTP-Binding Protein alpha Subunits, Gs