The von Hippel-Lindau tumor suppressor targets to mitochondria

Cancer Res. 2000 Jun 1;60(11):2816-9.

Abstract

Subcellular localization of von Hippel-Lindau (VHL) tumor suppressor may clarify its role in tumorigenesis. In rat kidney, we observed a granular cytoplasmic immunostaining of VHL, as seen in human tissues. The green fluorescent protein (GFP)-tagged VHL also appeared as cytoplasmic granules in vitro and was colocalized with a mitochondrion-selective dye. Immunogold electron microscopy localized VHL specifically to the mitochondrion. Mitochondria retaining GFP-VHL fusion protein, mimicking an insertional VHL mutant, displayed abnormal phenotypes. Among these, small mitochondria have been observed in clear cell renal carcinomas known to have frequent VHL alterations. Thus, VHL may contribute to tumorigenesis through mitochondria-based action.

Publication types

  • Research Support, U.S. Gov't, P.H.S.

MeSH terms

  • Animals
  • Cell Line
  • Cytoplasm / metabolism
  • Epithelial Cells / cytology
  • Epithelial Cells / metabolism
  • Green Fluorescent Proteins
  • Immunohistochemistry
  • Kidney / metabolism
  • Ligases*
  • Luminescent Proteins / metabolism
  • Microscopy, Confocal
  • Mitochondria / metabolism*
  • Plasmids
  • Protein Biosynthesis*
  • Rats
  • Transfection
  • Tumor Suppressor Proteins*
  • Ubiquitin-Protein Ligases*
  • Von Hippel-Lindau Tumor Suppressor Protein

Substances

  • Luminescent Proteins
  • Tumor Suppressor Proteins
  • Green Fluorescent Proteins
  • Ubiquitin-Protein Ligases
  • Von Hippel-Lindau Tumor Suppressor Protein
  • Ligases