The diuretic- and Bartter-like salt-losing tubulopathies

Nephrol Dial Transplant. 2000;15 Suppl 6:19-20. doi: 10.1093/ndt/15.suppl_6.19.
No abstract available

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Anion Transport Proteins*
  • Bartter Syndrome / metabolism*
  • Benzothiadiazines
  • Chloride Channels / genetics
  • Diuretics / adverse effects*
  • Furosemide / adverse effects
  • Heterozygote
  • Homozygote
  • Humans
  • Infant, Newborn
  • Kidney Diseases / chemically induced*
  • Kidney Diseases / metabolism*
  • Kidney Tubules*
  • Membrane Proteins*
  • Mutation / physiology
  • Potassium Channels / genetics
  • Potassium Channels, Inwardly Rectifying*
  • Sodium Chloride / metabolism*
  • Sodium Chloride Symporter Inhibitors / adverse effects

Substances

  • Anion Transport Proteins
  • Benzothiadiazines
  • CLCNKB protein, human
  • Chloride Channels
  • Diuretics
  • KCNJ1 protein, human
  • Membrane Proteins
  • Potassium Channels
  • Potassium Channels, Inwardly Rectifying
  • Sodium Chloride Symporter Inhibitors
  • Sodium Chloride
  • Furosemide