Hypothesis: is a failure to prevent bacteriolysis and the synergy among microbial and host-derived pro-inflammatory agonists the main contributory factors to the pathogenesis of post-infectious sequelae?

Inflammation. 2001 Feb;25(1):1-6. doi: 10.1023/a:1007024809661.

Author

I Ginsburg¹

Affiliation

¹ Department of Oral Biology, Hebrew University-Hadassah Faculty of Dental Medicine, Jerusalem, Israel.

PMID: 11293661
DOI: 10.1023/a:1007024809661

No abstract available

Publication types

Research Support, Non-U.S. Gov't

MeSH terms

Animals
Anti-Bacterial Agents / pharmacology
Anti-Bacterial Agents / therapeutic use
Anti-Inflammatory Agents / administration & dosage
Anti-Inflammatory Agents / therapeutic use
Bacteremia / complications
Bacteremia / diagnosis
Bacteremia / drug therapy
Bacteriolysis* / drug effects
Cell Wall / drug effects
Cell Wall / metabolism
Critical Care
Culture
Drug Administration Schedule
Drug Therapy, Combination
Endotoxins / metabolism
Humans
Inflammation Mediators / physiology*
Lipopolysaccharides / metabolism
Models, Biological
Phagocytes / enzymology
Phagocytosis
Sepsis / drug therapy
Sepsis / physiopathology*
Sepsis / prevention & control

Substances

Anti-Bacterial Agents
Anti-Inflammatory Agents
Endotoxins
Inflammation Mediators
Lipopolysaccharides