Endomorphin-1 and endomorphin-2: pharmacology of the selective endogenous mu-opioid receptor agonists

Pharmacol Ther. 2000 Dec;88(3):437-63. doi: 10.1016/s0163-7258(00)00100-5.

Abstract

The recently discovered endogenous opioid peptides, endomorphins-1 and -2, appear to have properties consistent with neurotransmitter/neuromodulator actions in mammals. This review surveys the information gained so far from studies of different aspects of the endomorphins. Thus, the endomorphins have been found unequally in the brain; they are stored in neurons and axon terminals, with a heterogeneous distribution; they are released from synaptosomes by depolarization; they are enzymatically converted by endopeptidases; and they interact specifically and with high affinity with mu-opioid receptors. The most outstanding effect of the endomorphins is their antinociceptive action. This depends on both central and peripheral neurons. Additionally, the endomorphins cause vasodilatation by stimulating nitric oxide release from the endothelium. Their roles in different central and peripheral functions, however, have not been fully clarified yet. From a therapeutic perspective, therefore, they may be conceived at present as potent antinociceptive and vasodilator agents.

Publication types

  • Research Support, Non-U.S. Gov't
  • Review

MeSH terms

  • Analgesics, Opioid / pharmacology*
  • Animals
  • Cardiovascular Physiological Phenomena
  • Digestive System Physiological Phenomena
  • Endothelium / physiology
  • Humans
  • Immune System
  • Inflammation
  • Mammals
  • Nitric Oxide
  • Oligopeptides / pharmacology*
  • Receptors, Opioid, mu / drug effects*
  • Receptors, Opioid, mu / physiology
  • Respiratory Physiological Phenomena
  • Signal Transduction
  • Vasodilation

Substances

  • Analgesics, Opioid
  • Oligopeptides
  • Receptors, Opioid, mu
  • endomorphin 1
  • Nitric Oxide
  • endomorphin 2