Regulation and execution of programmed cell death in response to pathogens, stress and developmental cues

Curr Opin Plant Biol. 2001 Dec;4(6):561-7. doi: 10.1016/s1369-5266(00)00216-8.

Abstract

Recent studies have expanded our view of the interactions between small molecule signals that regulate the hypersensitive response and other forms of cell suicide in plants. The mitochondrion has received increasing support as a mediator of at least some forms of programmed cell death in plants. In addition, new information provides a glimpse of how plant hormone signaling may be integrated with extensive autolysis, sensitivity to reactive oxygen intermediates and cell death.

Publication types

  • Research Support, Non-U.S. Gov't
  • Research Support, U.S. Gov't, Non-P.H.S.
  • Research Support, U.S. Gov't, P.H.S.
  • Review

MeSH terms

  • Adaptation, Physiological
  • Apoptosis / physiology*
  • Magnoliopsida / microbiology
  • Magnoliopsida / physiology*
  • Mitochondria / metabolism*
  • Reactive Oxygen Species / metabolism*
  • Signal Transduction

Substances

  • Reactive Oxygen Species