Saccadic peak velocity and EEG as end-points for a serotonergic challenge test

Hum Psychopharmacol. 2002 Mar;17(2):83-9. doi: 10.1002/hup.374.

Abstract

We previously reported that a single dose of the serotonin receptor agonist meta-chlorophenylpiperazine increased the peak velocity of saccadic eye movements and decreased low-frequency electroencephalographic activity.

Methods: We administered a single dose of the serotonin releaser dexfenfluramine in a double blind, placebo controlled randomised cross-over design and measured saccadic eye movements and EEG every hour up to 6 h. Subjects were 62 males (18-30 years) with a history of no, moderate or heavy use of ecstasy tablets.

Results: Dexfenfluramine increased saccadic peak velocity and decreased alpha, delta and theta electroencephalographic activity, the latter predominantly in heavy users of ecstasy.

Conclusions: This study supports the idea that saccadic peak velocity and EEG can be useful endpoints of a serotonergic challenge. This could be an important anatomical extension of these end-points, which until now were limited to the effect on hypothalamic serotonergic projections.

Publication types

  • Clinical Trial
  • Comparative Study
  • Randomized Controlled Trial
  • Research Support, Non-U.S. Gov't

MeSH terms

  • Adolescent
  • Adult
  • Cross-Over Studies
  • Dexfenfluramine / pharmacokinetics
  • Dexfenfluramine / pharmacology*
  • Dose-Response Relationship, Drug
  • Double-Blind Method
  • Electroencephalography
  • Hallucinogens
  • Humans
  • Male
  • N-Methyl-3,4-methylenedioxyamphetamine
  • Norfenfluramine / metabolism
  • Saccades / drug effects*
  • Serotonin / physiology*
  • Serotonin Receptor Agonists / pharmacokinetics
  • Serotonin Receptor Agonists / pharmacology*
  • Substance-Related Disorders / physiopathology

Substances

  • Hallucinogens
  • Serotonin Receptor Agonists
  • Norfenfluramine
  • Serotonin
  • Dexfenfluramine
  • N-Methyl-3,4-methylenedioxyamphetamine