Abstract
Specific binding of IGF-binding protein (IGFBP)-3 was shown to be present in the isolated, beating rat heart. The uptake of perfused (125)I-labeled IGF-I in the beating heart was decreased to 9% by blocking IGF-I binding sites with the IGF-I analog Long R(3) (LR(3)) IGF-I. When LR(3) was perfused with complexes of (125)I-IGF-I. IGFBP-3, uptake of (125)I-IGF-I was decreased to 41%, which was significantly greater than LR(3) and (125)I-IGF-I (41 vs. 9%). These data suggest that both microvessel IGF-I and IGFBP-3 binding sites contribute to the transport of IGF-I in the perfused rat heart. This also suggests a novel and plausible mechanism whereby circulating IGFs reach sites of IGF bioactivity.
Publication types
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Research Support, U.S. Gov't, Non-P.H.S.
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Research Support, U.S. Gov't, P.H.S.
MeSH terms
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Animals
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Autoradiography
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Binding Sites
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Biological Transport
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Cells, Cultured
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Endothelium, Vascular / metabolism
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Insulin-Like Growth Factor Binding Protein 3 / administration & dosage
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Insulin-Like Growth Factor Binding Protein 3 / genetics
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Insulin-Like Growth Factor Binding Protein 3 / metabolism*
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Insulin-Like Growth Factor Binding Protein 4 / genetics
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Insulin-Like Growth Factor Binding Protein 4 / metabolism
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Insulin-Like Growth Factor I / administration & dosage
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Insulin-Like Growth Factor I / metabolism*
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Iodine Radioisotopes
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Male
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Microcirculation / metabolism
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Myocardium / metabolism*
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Rats
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Rats, Sprague-Dawley
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Recombinant Fusion Proteins / metabolism
Substances
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Insulin-Like Growth Factor Binding Protein 3
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Insulin-Like Growth Factor Binding Protein 4
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Iodine Radioisotopes
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Recombinant Fusion Proteins
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Insulin-Like Growth Factor I