Abstract
Tin greatly enhances heme breakdown in kidney, thus impairing heme-dependent cellular functions, such as cytochrome P-450 mediated drug biotransformation. This novel action of the metal results from a potent induction effect on heme oxygenase, the enzyme that catalyzes heme oxidation in microsomes. The possible toxicological implications of this tin effect in the kidney merit further investigation.
MeSH terms
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5-Aminolevulinate Synthetase / biosynthesis
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Animals
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Cytochrome P-450 Enzyme System / metabolism
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Enzyme Induction / drug effects
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Heme / metabolism*
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Kidney / drug effects*
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Kidney / enzymology
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Kidney / ultrastructure
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Male
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Microsomes / enzymology
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Microsomes, Liver / enzymology
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Mixed Function Oxygenases / biosynthesis*
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Rats
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Tin / pharmacology*
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Tin / toxicity
Substances
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Heme
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Tin
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Cytochrome P-450 Enzyme System
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Mixed Function Oxygenases
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5-Aminolevulinate Synthetase