[Extracorporeal experimental study on immuno-modulatory activity of Astragalus memhranaceus extract]

Zhongguo Zhong Xi Yi Jie He Za Zhi. 2002 Jun;22(6):453-6.
[Article in Chinese]

Abstract

Objective: To study the effect of Astragalus membranaceus extract (AME) in regulating the immune function of human peripheral blood immune cells (PBIC) in vitro.

Methods: Effects of AME on the proliferation activity of peripheral blood mononuclear cells (PBMC) and the tumor cell phagocytosis of peripheral blood adherent monocytes (PBAM) were measured by using 3H-TdR incorporation. Effect of the tumor-killing activity of cytotoxic T-lymphocyte (CTL) was determined by using 51Cr-releasing assay. Effects on production of IgG by peripheral blood B cells (PBBC) and IL-6 by PBAM were tested by means of ELISA, and effect on production of TNF-alpha by PBAM was studied by means of biological method. Besides, the protein elements of AME were analysed by SDS-PAGE.

Results: AME could promote the proliferation of human PBMC, elevate the tumor cell-killing activity of CTL, strengthen the tumor cell phagocytosis and cytokines (TNF-alpha and IL-6) production of PBAM, and promote the IgG production of PBBC. AME contained multiple protein elements.

Conclusion: AME has effect in enhancing human immuno-function and anti-tumor activity, it could be applied in clinical practice for immuno-modulation and tumor treatment.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Adjuvants, Immunologic / pharmacology*
  • Antineoplastic Agents / pharmacology
  • Astragalus propinquus / chemistry*
  • Cytotoxicity, Immunologic / drug effects
  • Drugs, Chinese Herbal / pharmacology*
  • Humans
  • In Vitro Techniques
  • Interleukin-6 / blood
  • Leukocytes, Mononuclear / immunology*
  • Phagocytosis / drug effects
  • T-Lymphocytes, Cytotoxic / immunology
  • Tumor Necrosis Factor-alpha / metabolism

Substances

  • Adjuvants, Immunologic
  • Antineoplastic Agents
  • Drugs, Chinese Herbal
  • Interleukin-6
  • Tumor Necrosis Factor-alpha