Effect of granulocyte-macrophage colony-stimulating factor on natural-killer cell mediated cytotoxicity

Int J Biochem Cell Biol. 2003 Jul;35(7):1056-60. doi: 10.1016/s1357-2725(02)00383-7.

Abstract

Previous studies demonstrated an enhancing effect of granulocyte-macrophage colony-stimulating-factor (GM-CSF) on natural cytotoxicity. It was the aim of this study to investigate if CD56(+) natural-killer (NK) cells are responsible for the increased natural cytotoxicity after GM-CSF treatment. NK-cells were incubated with or without GM-CSF and Interferon-alpha (IFN-alpha) at various concentrations. NK-activity was determined by their ability to lyse NK-sensitive tumor cells (K562) and by cell surface expression of activation markers (CD25 and CD69). In our experimental setting incubation of CD56(+) NK-cells with GM-CSF did not significantly alter NK-cell mediated cytotoxicity or the expression of activation markers. In contrast, pre-treatment with IFN-alpha, a well known stimulant of NK-activity enhanced cytotoxicity by 69.2%+/-13.2%, P<0.05, effector/target cell ratio (E/T) 10:1 and by 43.3%+/-17.3%, P<0.05, E/T 20:1 and increased the expression of CD69 and CD25. Our results suggest that GM-CSF treatment alone cannot enhance natural cytotoxicity mediated by CD56(+) NK-cells in vitro.

MeSH terms

  • Antineoplastic Agents / pharmacology
  • CD56 Antigen / immunology
  • Cells, Cultured
  • Cytotoxicity, Immunologic*
  • Granulocyte-Macrophage Colony-Stimulating Factor / immunology*
  • Granulocyte-Macrophage Colony-Stimulating Factor / pharmacology
  • Humans
  • Interferon alpha-2
  • Interferon-alpha / pharmacology
  • K562 Cells
  • Killer Cells, Natural / drug effects
  • Killer Cells, Natural / immunology*
  • Recombinant Proteins
  • T-Lymphocyte Subsets / drug effects
  • T-Lymphocyte Subsets / immunology*

Substances

  • Antineoplastic Agents
  • CD56 Antigen
  • Interferon alpha-2
  • Interferon-alpha
  • Recombinant Proteins
  • Granulocyte-Macrophage Colony-Stimulating Factor