Abstract
The senses of hearing and balance in vertebrates rely on the sensory hair cells (HCs) of the inner ear. The central element of the HC's transduction apparatus is a mechanically gated ion channel of unknown identity. Here we report that the zebrafish ortholog of Drosophila no mechanoreceptor potential C (nompC), which encodes a transient receptor potential (TRP) channel, is critical for HC mechanotransduction. In zebrafish larvae, nompC is selectively expressed in sensory HCs. Morpholino-mediated removal of nompC function eliminated transduction-dependent endocytosis and electrical responses in HCs, resulting in larval deafness and imbalance. These observations indicate that nompC encodes a vertebrate HC mechanotransduction channel.
Publication types
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Research Support, Non-U.S. Gov't
MeSH terms
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Amino Acid Sequence
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Animals
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Cochlear Microphonic Potentials
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Computational Biology
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Deafness
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Ear, Inner / embryology
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Endocytosis
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Gene Expression
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Hair Cells, Auditory / physiology*
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Hearing
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In Situ Hybridization
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Ion Channels / chemistry
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Ion Channels / genetics
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Ion Channels / physiology*
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Mechanotransduction, Cellular*
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Molecular Sequence Data
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Oligonucleotides, Antisense
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Phenotype
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Phylogeny
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Postural Balance
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Reflex, Startle
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Reverse Transcriptase Polymerase Chain Reaction
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Transient Receptor Potential Channels
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Zebrafish
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Zebrafish Proteins / chemistry
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Zebrafish Proteins / genetics
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Zebrafish Proteins / physiology*
Substances
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Ion Channels
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Oligonucleotides, Antisense
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Transient Receptor Potential Channels
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Zebrafish Proteins
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trpn1 protein, zebrafish