Involvement of GABA-dependent chloride channel in the action of anticonvulsant and convulsant drugs

Epilepsy Res Suppl. 1992:8:77-85. doi: 10.1016/b978-0-444-89710-7.50016-1.
No abstract available

MeSH terms

  • Animals
  • Anticonvulsants / pharmacology*
  • Bridged Bicyclo Compounds / pharmacokinetics
  • Bridged Bicyclo Compounds, Heterocyclic*
  • Cerebral Cortex / drug effects
  • Cerebral Cortex / physiopathology
  • Chloride Channels
  • Convulsants / pharmacokinetics
  • Convulsants / pharmacology*
  • Culture Techniques
  • Epilepsy / chemically induced*
  • Epilepsy / physiopathology
  • Isoniazid / pharmacology
  • Male
  • Membrane Proteins / drug effects*
  • Membrane Proteins / physiology
  • Rats
  • Rats, Sprague-Dawley
  • Receptors, GABA-A / drug effects*
  • Receptors, GABA-A / physiology
  • Synapses / drug effects
  • Synapses / physiology
  • Synaptic Transmission / drug effects*
  • Synaptic Transmission / physiology
  • Valproic Acid / pharmacology
  • gamma-Aminobutyric Acid / physiology*

Substances

  • Anticonvulsants
  • Bridged Bicyclo Compounds
  • Bridged Bicyclo Compounds, Heterocyclic
  • Chloride Channels
  • Convulsants
  • Membrane Proteins
  • Receptors, GABA-A
  • gamma-Aminobutyric Acid
  • Valproic Acid
  • tert-butylbicyclophosphorothionate
  • Isoniazid