Transcriptional activation of jun and actin genes by estrogen during mitogenic stimulation of rat uterine cells

J Steroid Biochem Mol Biol. 1992 Mar;41(3-8):523-8. doi: 10.1016/0960-0760(92)90377-u.

Abstract

Estrogens induce transcriptional activation of c-fos and c-myc proto-oncogenes during mitogenic stimulation of human, chicken, mouse and rat cells in vivo and in vitro. In this paper we show that 17 beta-estradiol injected into adult ovariectomized rats increases c-jun, jun-B and jun-D gene transcription in the uterus. Kinetics and amplitude of response are different for each gene, since c-jun is activated first, within 30 min after injection, followed by jun-D and jun-B, 60 and 90 min after injection, respectively. Maximal activation of jun-B marks a drop in transcription of all the jun genes. Furthermore, transcriptional activation by 17 beta-estradiol of the growth-regulated beta- and gamma-cytoskeletal actin genes is prevented by an inhibitor of protein synthesis, indicating that it is a secondary response to the hormone. These data support the hypothesis that during growth stimulation of target cells the estrogen receptor induces transcription of regulatory genes, triggering in this way a cascade of gene regulation events that results in progression through the cell cycle.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Actins / genetics*
  • Animals
  • Cell Nucleus / drug effects
  • Cell Nucleus / metabolism*
  • DNA Probes
  • Estradiol / pharmacology*
  • Female
  • Gene Expression Regulation* / drug effects
  • Genes, fos
  • Genes, jun* / drug effects
  • Genes, myc
  • Kinetics
  • Ovariectomy
  • Poly A / genetics
  • Poly A / isolation & purification
  • RNA / genetics
  • RNA / isolation & purification
  • RNA, Messenger / genetics
  • RNA, Messenger / metabolism
  • Rats
  • Rats, Inbred Strains
  • Transcription, Genetic* / drug effects
  • Uterus / drug effects
  • Uterus / metabolism*

Substances

  • Actins
  • DNA Probes
  • RNA, Messenger
  • Poly A
  • Estradiol
  • RNA