Abstract
In this study we show that both glycogen synthase kinase 3 (GSK3) isoforms, GSK3alpha and GSK3beta, are present in human platelets and are phosphorylated on Ser(21) and Ser(9), respectively, in platelets stimulated with collagen, convulxin and thrombin. Phosphorylation of GSK3alpha/beta was dependent on phosphoinositide 3-kinase (PI3K) activity and independent of platelet aggregation, and correlated with a decrease in GSK3 activity that was preserved by pre-incubating platelets with PI3K inhibitor LY294002. Three structurally distinct GSK3 inhibitors, lithium, SB415286 and TDZD-8, were found to inhibit platelet aggregation. This implicates GSK3 as a potential regulator of platelet function.
Publication types
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Research Support, Non-U.S. Gov't
MeSH terms
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Blood Platelets / drug effects
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Blood Platelets / enzymology*
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Blood Platelets / physiology*
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Collagen / pharmacology
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Crotalid Venoms / pharmacology
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Enzyme Inhibitors / pharmacology
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Glycogen Synthase Kinase 3 / metabolism*
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Glycogen Synthase Kinase 3 beta
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Humans
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Lectins, C-Type*
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Phosphatidylinositol 3-Kinases / metabolism
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Phosphorylation / drug effects
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Platelet Activation / drug effects
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Platelet Aggregation / drug effects
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Protein Isoforms / metabolism
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Serine / metabolism
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Thrombin / pharmacology
Substances
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Crotalid Venoms
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Enzyme Inhibitors
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Lectins, C-Type
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Protein Isoforms
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convulxin
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Serine
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Collagen
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Phosphatidylinositol 3-Kinases
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GSK3B protein, human
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Glycogen Synthase Kinase 3 beta
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Glycogen Synthase Kinase 3
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glycogen synthase kinase 3 alpha
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Thrombin