Folic acid rescues nitric oxide-induced neural tube closure defects

Cell Death Differ. 2004 Mar;11(3):361-3. doi: 10.1038/sj.cdd.4401371.
No abstract available

Publication types

  • Letter
  • Research Support, Non-U.S. Gov't

MeSH terms

  • 5-Methyltetrahydrofolate-Homocysteine S-Methyltransferase / antagonists & inhibitors
  • 5-Methyltetrahydrofolate-Homocysteine S-Methyltransferase / drug effects
  • Animals
  • Carbon Radioisotopes
  • Chick Embryo
  • Culture Techniques / methods
  • Folic Acid / metabolism*
  • Folic Acid / physiology
  • Hemoglobins / metabolism
  • Homocysteine / metabolism
  • Models, Biological
  • Neural Tube Defects / chemically induced
  • Neural Tube Defects / embryology
  • Neural Tube Defects / prevention & control*
  • Neural Tube Defects / ultrastructure
  • Nitric Oxide / metabolism*
  • Nitric Oxide / pharmacology*
  • Nitric Oxide Donors / pharmacology
  • Penicillamine / analogs & derivatives*
  • Penicillamine / metabolism
  • Penicillamine / pharmacology*
  • Tetrahydrofolates / metabolism
  • Time Factors

Substances

  • Carbon Radioisotopes
  • Hemoglobins
  • Nitric Oxide Donors
  • S-nitro-N-acetylpenicillamine
  • Tetrahydrofolates
  • Homocysteine
  • Nitric Oxide
  • Folic Acid
  • 5-Methyltetrahydrofolate-Homocysteine S-Methyltransferase
  • Penicillamine
  • 5-methyltetrahydrofolate