Apoptosis in liver damage produced by tunicamycin

Aust Vet J. 2004 Jan-Feb;82(1-2):87-90. doi: 10.1111/j.1751-0813.2004.tb14652.x.

Abstract

Objective: To determine whether apoptosis contributes to hepatocyte loss in tunicamycin poisoning.

Design: Groups of four guinea pigs were given 400 microg/kg of tunicamycin subcutaneously and killed at 24 h intervals up to 72 h post-injection. Livers were examined by routine histological methods and ancillary techniques (TUNEL staining, endonuclease activation, caspase activity, and electron microscopy) to ascertain whether any hepatocyte injury was apoptotic.

Results: Many hepatocytes exposed to tunicamycin showed evidence of apoptosis in the form of nuclear karyorrhexis with chromatin margination and crescent formation, TUNEL-positivity, DNA laddering, elevated caspase activity and apoptotic body formation.

Conclusion: Tunicamycin caused the death of many hepatocytes in the livers of guinea pigs by apoptosis.

MeSH terms

  • Animal Feed
  • Animals
  • Animals, Domestic
  • Apoptosis / drug effects
  • Guinea Pigs
  • In Situ Nick-End Labeling / veterinary
  • Injections, Subcutaneous
  • Liver / cytology
  • Liver / drug effects*
  • Liver / enzymology
  • Liver / pathology
  • Liver / ultrastructure
  • Plant Poisoning / pathology
  • Plant Poisoning / veterinary
  • Tunicamycin / poisoning*

Substances

  • Tunicamycin