Patterns of collateral pathways in extrahepatic portal hypertension as demonstrated by multidetector row computed tomography and advanced image processing

J Comput Assist Tomogr. 2004 Jul-Aug;28(4):469-77. doi: 10.1097/00004728-200407000-00006.

Abstract

Pancreatic neoplasm can compromise the portal vein or its tributaries, resulting in extrahepatic portal hypertension. Collateral channels may develop, usually in a hepatopetal direction. These include gastroepiploic veins, short gastric veins, left gastric vein, left colic veins, and spontaneous splenorenal shunts. We present the spectrum of these collaterals as delineated by multidetector row computed tomography with advanced image processing. Delineation of these collaterals is important before surgery so as to avoid possible uncontrollable bleeding if they are inadvertently disrupted.

MeSH terms

  • Aged
  • Collateral Circulation / physiology*
  • Constriction, Pathologic / diagnostic imaging
  • Constriction, Pathologic / physiopathology
  • Female
  • Humans
  • Hypertension, Portal / diagnostic imaging
  • Hypertension, Portal / physiopathology*
  • Image Processing, Computer-Assisted / methods*
  • Male
  • Mesenteric Vascular Occlusion / diagnostic imaging
  • Mesenteric Vascular Occlusion / physiopathology
  • Mesenteric Veins / diagnostic imaging
  • Mesenteric Veins / physiopathology
  • Middle Aged
  • Pancreatic Neoplasms / diagnostic imaging
  • Peripheral Vascular Diseases / diagnostic imaging
  • Peripheral Vascular Diseases / physiopathology
  • Portal System / physiopathology
  • Portal Vein / physiopathology
  • Portography
  • Splenic Vein / diagnostic imaging
  • Splenic Vein / physiopathology
  • Tomography, X-Ray Computed / methods*