Changing patterns of pneumoconiosis mortality--United States, 1968-2000
- PMID: 15269698
Changing patterns of pneumoconiosis mortality--United States, 1968-2000
Abstract
Pneumoconioses are caused by the inhalation and deposition of mineral dusts in the lungs, resulting in pulmonary fibrosis and other parenchymal changes. Many persons with early pneumoconiosis are asymptomatic, but advanced disease often is accompanied by disability and premature death. Known pneumoconioses include coal workers' pneumoconiosis (CWP), silicosis, asbestosis, mixed dust pneumoconiosis, graphitosis, and talcosis. No effective treatment for these diseases is available. This report describes the temporal patterns of pneumoconiosis mortality during 1968-2000, which indicates an overall decrease in pneumoconiosis mortality. However, asbestosis increased steadily and is now the most frequently recorded pneumoconiosis on death certificates. Increased awareness of this trend is needed among health-care providers, employers, workers, and public health agencies.
Similar articles
-
Coal workers' pneumoconiosis-related years of potential life lost before age 65 years - United States, 1968-2006.MMWR Morb Mortal Wkly Rep. 2009 Dec 25;58(50):1412-6. MMWR Morb Mortal Wkly Rep. 2009. PMID: 20032926
-
Recent trends of age-specific pneumoconiosis mortality rates in the United States, 1985-1996: coal workers' pneumoconiosis, asbestosis, and silicosis.Int J Occup Environ Health. 1999 Oct-Dec;5(4):251-5. doi: 10.1179/oeh.1999.5.4.251. Int J Occup Environ Health. 1999. PMID: 10633240
-
Trends in Pneumoconiosis Deaths - United States, 1999-2018.MMWR Morb Mortal Wkly Rep. 2020 Jun 12;69(23):693-698. doi: 10.15585/mmwr.mm6923a1. MMWR Morb Mortal Wkly Rep. 2020. PMID: 32525855 Free PMC article.
-
Recent advances in the pathogenesis and clinical assessment of mineral dust pneumoconioses: asbestosis, silicosis and coal pneumoconiosis.Eur Respir J. 1989 Nov;2(10):988-1001. Eur Respir J. 1989. PMID: 2691279 Review.
-
Lung disease caused by exposure to coal mine and silica dust.Semin Respir Crit Care Med. 2008 Dec;29(6):651-61. doi: 10.1055/s-0028-1101275. Epub 2009 Feb 16. Semin Respir Crit Care Med. 2008. PMID: 19221963 Review.
Cited by
-
Impact of Epidemic Intelligence Service Training in Occupational Respiratory Epidemiology.ATS Sch. 2023 Sep 20;4(4):441-463. doi: 10.34197/ats-scholar.2023-0062PS. eCollection 2023 Dec. ATS Sch. 2023. PMID: 38196681 Free PMC article.
-
Increased odds of mortality from non-malignant respiratory disease and lung cancer are highest among US coal miners born after 1939.Occup Environ Med. 2023 Mar;80(3):121-128. doi: 10.1136/oemed-2022-108539. Epub 2023 Jan 12. Occup Environ Med. 2023. PMID: 36635098 Free PMC article.
-
Characteristics of Diagnosed and Death Cases of Pneumoconiosis in Hubei Province, China, 1949-2019.Int J Environ Res Public Health. 2022 Nov 27;19(23):15799. doi: 10.3390/ijerph192315799. Int J Environ Res Public Health. 2022. PMID: 36497874 Free PMC article.
-
Demonstrating the protective effect of a 70-year-old occupational exposure limit against pneumoconiosis caused by mica.Toxicol Ind Health. 2022 Feb;38(2):63-69. doi: 10.1177/07482337211062281. Epub 2022 Mar 1. Toxicol Ind Health. 2022. PMID: 35230205 Free PMC article.
-
NOX4 regulates macrophage apoptosis resistance to induce fibrotic progression.J Biol Chem. 2021 Jul;297(1):100810. doi: 10.1016/j.jbc.2021.100810. Epub 2021 May 21. J Biol Chem. 2021. PMID: 34023385 Free PMC article.
MeSH terms
LinkOut - more resources
Full Text Sources
Molecular Biology Databases

