Clinical perspectives: neuroprotection lessons from hypoxia-tolerant organisms

J Exp Biol. 2004 Aug;207(Pt 18):3243-9. doi: 10.1242/jeb.00977.

Abstract

An effective treatment for brain ischemia is a pressing medical need. Research on brain ischemia has largely focused on understanding the mechanisms of neuron death as a way of identifying targets for therapy. An attractive alternative approach is to identify the survival strategies of hypoxia-tolerant neurons. The adaptation of vertebrate neurons to hypoxia occurs in at least three major ways: (1) as a constitutive property of neurons in anoxia-tolerant turtles and fish, (2) as a property of intra-uterine and early post-natal mammalian development, and (3) as part of a slower, chronic process, as in acclimitization to high altitude. Research on hypoxia-tolerant neurons has already revised several earlier concepts, including the role of calcium in cell death and survival, and the value of N-methyl-d-aspartate (NMDA) receptor antagonism. A broad and fundamental understanding of how neurons adapt to hypoxia is likely to help guide efforts to find new treatments for brain hypoxia and ischemia.

Publication types

  • Research Support, U.S. Gov't, P.H.S.
  • Review

MeSH terms

  • Acclimatization / physiology
  • Adaptation, Physiological*
  • Altitude
  • Animals
  • Apoptosis / physiology
  • Brain Ischemia / metabolism
  • Brain Ischemia / physiopathology*
  • Calcium / metabolism
  • Fishes / metabolism*
  • Mammals / embryology
  • Mammals / metabolism*
  • Models, Biological
  • Neurons / metabolism*
  • Oxygen / metabolism
  • Receptors, N-Methyl-D-Aspartate / metabolism
  • Turtles / metabolism*

Substances

  • Receptors, N-Methyl-D-Aspartate
  • Oxygen
  • Calcium