Ultraviolet B-induced DNA damage in human epidermis is modified by the antioxidants ascorbic acid and D-alpha-tocopherol

J Invest Dermatol. 2005 Feb;124(2):304-7. doi: 10.1111/j.0022-202X.2004.23560.x.

Abstract

DNA damage caused by ultraviolet (UV) irradiation is considered the main etiologic factor contributing to the development of skin cancer. Systemic or topical application of antioxidants has been suggested as a protective measure against UV-induced skin damage. We investigated the effect of long-term oral administration of a combination of the antioxidants ascorbic acid (vitamin C) and D-alpha-tocopherol (vitamin E) in human volunteers on UVB-induced epidermal damage. The intake of vitamins C and E for a period of 3 mo significantly reduced the sunburn reaction to UVB irradiation. Detection of thymine dimers in the skin using a specific antibody revealed a significant increase of this type of DNA damage following UVB exposure. After 3 mo of antioxidant administration, significantly less thymine dimers were induced by the UVB challenge, suggesting that antioxidant treatment protected against DNA damage.

Publication types

  • Clinical Trial
  • Research Support, Non-U.S. Gov't

MeSH terms

  • Administration, Oral
  • Adult
  • Aged
  • Antioxidants / administration & dosage*
  • Ascorbic Acid / administration & dosage*
  • Biopsy
  • DNA Damage / drug effects
  • Drug Therapy, Combination
  • Epidermis / drug effects*
  • Epidermis / metabolism
  • Epidermis / radiation effects
  • Female
  • Humans
  • Male
  • Middle Aged
  • Pyrimidine Dimers / metabolism
  • Sunburn / drug therapy*
  • Sunburn / metabolism
  • Ultraviolet Rays
  • alpha-Tocopherol / administration & dosage*

Substances

  • Antioxidants
  • Pyrimidine Dimers
  • alpha-Tocopherol
  • Ascorbic Acid