Biochemistry: a pore way to die

Nature. 2005 Mar 31;434(7033):578-9. doi: 10.1038/434578a.
No abstract available

Publication types

  • News
  • Comment

MeSH terms

  • Animals
  • Apoptosis / drug effects
  • Calcium / metabolism
  • Calcium / pharmacology
  • Cyclophilin D
  • Cyclophilins / deficiency
  • Cyclophilins / genetics
  • Cyclophilins / metabolism*
  • Mice
  • Mitochondria / drug effects
  • Mitochondria / genetics
  • Mitochondria / metabolism*
  • Mitochondria / pathology
  • Mitochondrial ADP, ATP Translocases / chemistry
  • Mitochondrial ADP, ATP Translocases / metabolism
  • Mitochondrial Swelling / drug effects
  • Mitochondrial Swelling / physiology
  • Myocardial Ischemia / genetics
  • Myocardial Ischemia / metabolism
  • Myocardial Ischemia / pathology
  • Myocardial Reperfusion Injury / genetics
  • Myocardial Reperfusion Injury / metabolism
  • Myocardial Reperfusion Injury / pathology
  • Myocytes, Cardiac / cytology
  • Myocytes, Cardiac / drug effects
  • Myocytes, Cardiac / metabolism
  • Myocytes, Cardiac / pathology
  • Necrosis*
  • Oxidative Stress

Substances

  • Cyclophilin D
  • PPIF protein, mouse
  • Mitochondrial ADP, ATP Translocases
  • Cyclophilins
  • Calcium