A short duration transient ischemia induces apoptosis in retinal layers: an experimental study in rabbits

Eur J Ophthalmol. 2005 Mar-Apr;15(2):233-8. doi: 10.1177/112067210501500210.

Abstract

Purpose: To investigate retinal cell apoptosis in an experimental transient, short duration ocular ischemia model.

Methods: An experimental ischemia model, which simulates creating temporary high intraocular pressure to control intraocular bleeding during pars plana vitrectomy, was set up. Rabbits were randomly divided into three groups. Group 1 was the control group. In Group 2, intraocular pressure was increased to 97 mmHg for 5 minutes. In Group 3, intraocular pressure was increased to 97 mmHg for 10 minutes. After 24 hours, terminal deoxyribonucleotidyl transferase-mediated dUTP-digoxigenin nick-end labeling assay was used to detect retinal apoptosis in rabbit eyes. Only nuclear staining in retinal cells was counted.

Results: Groups with 5 minutes and 10 minutes of ischemia showed significantly higher amount of ganglion cell layer apoptosis when compared with the control group (p<0.05). Light microscopy and standard hematoxylin-eosin did not show any significant damage in the retina cells.

Conclusions: Apoptotic cell death in the retinal cell layers occurs in temporary ischemia-reperfusion as early as 5 and 10 minutes duration.

MeSH terms

  • Animals
  • Apoptosis*
  • Disease Models, Animal
  • In Situ Nick-End Labeling
  • Intraocular Pressure
  • Ocular Hypertension / complications
  • Rabbits
  • Reperfusion Injury / diagnosis*
  • Reperfusion Injury / etiology
  • Retinal Artery / pathology*
  • Retinal Diseases / diagnosis*
  • Retinal Diseases / etiology
  • Retinal Ganglion Cells / pathology*