Outlasting excitability shifts induced by direct current stimulation of the human brain

Suppl Clin Neurophysiol. 2004:57:708-14. doi: 10.1016/s1567-424x(09)70411-8.

Abstract

tDCS appears to be a promising tool in neuroplasticity research with some perspectives in clinical neurophysiology. It is closely related to modulation of cortical excitability and activity which are key mechanisms for modulating neuroplasticity. Long-term potentiation and long-term depression-like effects have been shown to be involved in learning processes in animal studies so far. Stimulation with weak direct currents is capable of inducing stimulation-polarity-dependent, prolonged, diminutions or elevations of cortical activity and excitability, most probably elicited by a hyper- or depolarisation of resting membrane potentials. Moreover, these modulations are functionally important, since they affect learning processes and epileptic activity. Here excitability changes have been accomplished in the human by non-invasive transcranial direct current stimulation (tDCS). They share some important features with these well-known neuroplastic changes: The duration of the effects depends on stimulation duration and intensity, they are of intracortical origin, and the prolonged effects depend on NMDA-receptor activity. Thus, this technique is a promising method in the field of neuroplastic research in animals and humans and could evolve as a therapeutic tool in some neuro-psychiatric disorders which benefit from modulation of cortical excitability.

Publication types

  • Review

MeSH terms

  • Brain / drug effects
  • Brain / physiology
  • Brain / radiation effects*
  • Calcium / metabolism
  • Calcium Channel Blockers / pharmacology
  • Dose-Response Relationship, Radiation
  • Electric Stimulation / methods*
  • Electrodes
  • Evoked Potentials, Motor / drug effects
  • Evoked Potentials, Motor / physiology
  • Evoked Potentials, Motor / radiation effects*
  • Excitatory Amino Acid Antagonists / pharmacology
  • Humans
  • Neural Inhibition
  • Neuronal Plasticity / drug effects
  • Neuronal Plasticity / physiology
  • Neuronal Plasticity / radiation effects*
  • Psychophysics / methods
  • Receptors, N-Methyl-D-Aspartate / physiology
  • Time Factors

Substances

  • Calcium Channel Blockers
  • Excitatory Amino Acid Antagonists
  • Receptors, N-Methyl-D-Aspartate
  • Calcium