Effects of lead on pH and temperature-dependent substrate-activation kinetics of ATPase system and its protection by thiol compounds in rat brain

Biomed Environ Sci. 1991 Dec;4(4):441-51.

Abstract

Lead (Pb) inhibited the activities of Na(+)-K+ ATPase (IC50 = 2.0 x 10(-6) M), K(+)-Para-Nitrophenyl phosphatase (PNPPase) (IC50 = 3.5 x 10(-6) M) and [3H]-ouabain binding (IC50 = 4.0 x 10(-5) M) in rat brain P2 fraction. A variable temperature or pH significantly elevated the inhibition of Na(+)-K+ ATPase by Pb in buffered acidic, neutral and alkaline pH ranges. Noncompetitive inhibition with respect to activation of Na(+)-K+ ATPase by ATP was indicated by a variation in Vmax values with no significant changes in Km values at any temperature studied. In the presence of Pb, for Na(+)-K+ ATPase at pH 6.5 and 8.5, Vmax was decreased with an increase in Km values suggesting a mixed type of inhibition. Sulfhydryl agents such as dithiothreitol (DTT) and cysteine (Cyst), but not glutathione (GSH) offered varied levels of protection against Pb-inhibition of Na(+)-K+ ATPase at pH 7.5 and 8.5. The present data suggest that inhibition of Na(+)-K+ ATPase by Pb is both temperature and pH-dependent. These results also indicate that Pb inhibited Na(+)-K+ ATPase by interfering with phosphorylation of enzyme molecule and dephosphorylation of the enzyme-phosphoryl complex and exerted an effect similar to that of SH-blocking agents.

Publication types

  • Research Support, U.S. Gov't, P.H.S.

MeSH terms

  • 4-Nitrophenylphosphatase / metabolism
  • Adenosine Triphosphate / metabolism
  • Animals
  • Brain / drug effects*
  • Brain / enzymology
  • Brain / metabolism
  • Enzyme Activation
  • Hydrogen-Ion Concentration
  • Kinetics
  • Lead / toxicity*
  • Male
  • Ouabain / metabolism
  • Rats
  • Rats, Inbred Strains
  • Sodium-Potassium-Exchanging ATPase / metabolism*
  • Substrate Specificity
  • Sulfhydryl Compounds / pharmacology*
  • Temperature

Substances

  • Sulfhydryl Compounds
  • Lead
  • Ouabain
  • Adenosine Triphosphate
  • 4-Nitrophenylphosphatase
  • Sodium-Potassium-Exchanging ATPase