Increased phosphorylation of caveolin-1 in the spinal cord of Lewis rats with experimental autoimmune encephalomyelitis

Neurosci Lett. 2006 Jul 10;402(1-2):76-80. doi: 10.1016/j.neulet.2006.04.008. Epub 2006 May 5.

Abstract

The expression of phospho-specific caveolin-1 (p-caveolin-1) was analyzed in the spinal cord of Lewis rats with experimental autoimmune encephalomyelitis (EAE). Western blot analysis showed that p-caveolin-1 was constitutively expressed in normal spinal cords and that it significantly increased in the spinal cord with EAE at both early and peak stages of EAE (P<0.05), and decreased slightly at the recovery stage of EAE. Immunohistochemistry showed that p-caveolin-1 was constitutively expressed in few vascular endothelial cells and glial cells in the spinal cords of normal rats. In EAE lesions, p-caveolin-1 was intensely immunostained in inflammatory T cells and macrophages. Therefore, we postulate that phosphorylation of caveolin-1 occurred in the inflammatory cells of EAE lesions, and that caveolin-associated cell activation is mainly associated with inflammatory cells that appear during early and peak stages of EAE.

Publication types

  • Comparative Study
  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Blotting, Western / methods
  • Caveolin 1 / metabolism*
  • Ectodysplasins / metabolism
  • Encephalomyelitis, Autoimmune, Experimental / pathology*
  • Female
  • Glial Fibrillary Acidic Protein / metabolism
  • Immunohistochemistry / methods
  • Lectins / metabolism
  • Phosphorylation
  • Rats
  • Rats, Inbred Lew
  • Spinal Cord / metabolism*
  • Time Factors

Substances

  • Caveolin 1
  • Ectodysplasins
  • Glial Fibrillary Acidic Protein
  • Lectins