Neovascularization in human atherosclerosis

Circulation. 2006 May 9;113(18):2245-52. doi: 10.1161/CIRCULATIONAHA.105.578955.
No abstract available

Publication types

  • Research Support, Non-U.S. Gov't
  • Review

MeSH terms

  • Angiotensin II / physiology
  • Animals
  • Aryl Hydrocarbon Receptor Nuclear Translocator / physiology
  • Atherosclerosis / pathology
  • Atherosclerosis / physiopathology*
  • Blood Proteins / metabolism
  • Capillary Permeability
  • Cell Hypoxia
  • Chemotaxis, Leukocyte
  • Coronary Artery Disease / pathology
  • Coronary Artery Disease / physiopathology
  • Coronary Vessels / physiopathology
  • Coronary Vessels / ultrastructure
  • Diagnostic Imaging
  • Disease Progression
  • Hemorrhage / etiology
  • Humans
  • Hypoxia-Inducible Factor 1, alpha Subunit / physiology
  • Macrophage Activation
  • Membrane Lipids / metabolism
  • Models, Animal
  • Neovascularization, Physiologic / physiology*
  • Oxidative Stress
  • Sus scrofa
  • Sympathetic Nervous System / physiopathology
  • Tunica Intima / pathology
  • Vasa Vasorum / physiopathology
  • Vascular Endothelial Growth Factor A / physiology

Substances

  • ARNT protein, human
  • Blood Proteins
  • HIF1A protein, human
  • Hypoxia-Inducible Factor 1, alpha Subunit
  • Membrane Lipids
  • VEGFA protein, human
  • Vascular Endothelial Growth Factor A
  • Angiotensin II
  • Aryl Hydrocarbon Receptor Nuclear Translocator