Copper homeostasis and neurodegenerative disorders (Alzheimer's, prion, and Parkinson's diseases and amyotrophic lateral sclerosis)

Chem Rev. 2006 Jun;106(6):1995-2044. doi: 10.1021/cr040410w.
No abstract available

Publication types

  • Review

MeSH terms

  • Adenosine Triphosphatases / chemistry
  • Adenosine Triphosphatases / metabolism
  • Alzheimer Disease / metabolism*
  • Alzheimer Disease / pathology
  • Amyloid beta-Peptides / chemistry
  • Amyloid beta-Peptides / metabolism
  • Amyotrophic Lateral Sclerosis / metabolism*
  • Amyotrophic Lateral Sclerosis / pathology
  • Animals
  • Copper* / chemistry
  • Copper* / metabolism
  • Histidine / chemistry
  • Histidine / metabolism
  • Homeostasis
  • Humans
  • Mice
  • Models, Chemical
  • Models, Molecular
  • Molecular Conformation
  • Molecular Structure
  • Neurodegenerative Diseases / metabolism
  • Neurodegenerative Diseases / pathology
  • Oxidative Stress
  • Parkinson Disease / metabolism*
  • Parkinson Disease / pathology
  • Prion Diseases / metabolism*
  • Prion Diseases / pathology
  • Protein Binding

Substances

  • Amyloid beta-Peptides
  • Histidine
  • Copper
  • Adenosine Triphosphatases