Responses of rod bipolar cells isolated from the rat retina to the glutamate agonist 2-amino-4-phosphonobutyric acid (APB)

J Neurosci. 1991 Aug;11(8):2372-82. doi: 10.1523/JNEUROSCI.11-08-02372.1991.

Abstract

Isolated rod bipolar cells were obtained by enzymatic (papain) and mechanical dissociation of the adult rat retina. Virtually all intact bipolar cells in the dissociates expressed protein kinase C (PKC) immunoreactivity, a selective marker for rod bipolar cells in the in vivo retina. Whole-cell recordings were performed using nystatin in the patch pipette to minimize washout of those cytoplasmic components necessary for the maintenance of ionic currents. At holding potentials of -33 mV, a tonic inward current was observed. The glutamate agonist 2-amino-4-phosphonobutyrate (APB) reduced this current by closing ion channels. Under normal conditions, Na+ appeared to be the main charge carrier. Both the internal and the external Ca2+ concentrations were found to exert a powerful influence on the APB-sensitive current. We conclude that the rod bipolar cell in situ is depolarized at light onset.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Aminobutyrates / pharmacology*
  • Animals
  • Calcium / pharmacology
  • Calcium / physiology
  • Carrier Proteins / physiology
  • Cell Separation
  • Electric Conductivity
  • Electrophysiology
  • Glutamates / physiology*
  • Glutamic Acid
  • Ion Channels / drug effects
  • Ion Channels / metabolism
  • Photoreceptor Cells / cytology
  • Photoreceptor Cells / drug effects*
  • Photoreceptor Cells / physiology
  • Protein Kinase C / physiology
  • Rats
  • Sodium-Calcium Exchanger

Substances

  • Aminobutyrates
  • Carrier Proteins
  • Glutamates
  • Ion Channels
  • Sodium-Calcium Exchanger
  • Glutamic Acid
  • Protein Kinase C
  • 2-amino-4-phosphonobutyric acid
  • Calcium