Hypothesis: antigen-specific T cells prime central nervous system endothelium for recruitment of nonspecific inflammatory cells to effect autoimmune demyelination

J Neuroimmunol. 1991 Sep;33(3):237-44. doi: 10.1016/0165-5728(91)90111-j.
No abstract available

Publication types

  • Research Support, Non-U.S. Gov't
  • Research Support, U.S. Gov't, P.H.S.
  • Review

MeSH terms

  • Animals
  • Antigens / immunology*
  • Autoimmune Diseases / pathology
  • Autoimmune Diseases / physiopathology*
  • Central Nervous System / blood supply*
  • Central Nervous System Diseases / pathology
  • Central Nervous System Diseases / physiopathology*
  • Demyelinating Diseases / pathology
  • Demyelinating Diseases / physiopathology*
  • Endothelium, Vascular / physiopathology*
  • Epitopes
  • Inflammation / pathology
  • Inflammation / physiopathology
  • Mice
  • T-Lymphocytes / immunology
  • T-Lymphocytes / physiology*

Substances

  • Antigens
  • Epitopes