Reversible glutathiolation of caspase-3 by glutaredoxin as a novel redox signaling mechanism in tumor necrosis factor-alpha-induced cell death

Circ Res. 2007 Feb 2;100(2):152-4. doi: 10.1161/01.RES.0000258171.08020.72.
No abstract available

Publication types

  • Comment
  • Editorial
  • Research Support, N.I.H., Extramural
  • Research Support, Non-U.S. Gov't
  • Review

MeSH terms

  • Animals
  • Apoptosis / physiology*
  • Caspase 3 / metabolism*
  • Caspase 3 / physiology
  • Caspase 8 / metabolism
  • Enzyme Precursors / metabolism*
  • Enzyme Precursors / physiology
  • Glutaredoxins
  • Glutathione / metabolism*
  • Humans
  • JNK Mitogen-Activated Protein Kinases / metabolism
  • MAP Kinase Signaling System / physiology
  • NF-kappa B / metabolism
  • Oxidation-Reduction
  • Oxidoreductases / physiology*
  • Signal Transduction / physiology*
  • Tumor Necrosis Factor-alpha / physiology*

Substances

  • Enzyme Precursors
  • Glutaredoxins
  • NF-kappa B
  • Tumor Necrosis Factor-alpha
  • Oxidoreductases
  • JNK Mitogen-Activated Protein Kinases
  • Caspase 3
  • Caspase 8
  • Glutathione