Response to bistability in apoptosis: roles of bax, bcl-2, and mitochondrial permeability transition pores

Biophys J. 2007 May 1;92(9):3332-4. doi: 10.1529/biophysj.106.100362. Epub 2007 Feb 2.
No abstract available

Publication types

  • Comment
  • Editorial
  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Apoptosis / physiology*
  • Cell Membrane Permeability / physiology*
  • Cell Survival
  • Computer Simulation
  • Humans
  • Ion Channels / metabolism*
  • Mitochondria / physiology*
  • Mitochondrial Membrane Transport Proteins / metabolism*
  • Models, Biological
  • Proto-Oncogene Proteins c-bcl-2 / metabolism*
  • bcl-2-Associated X Protein / metabolism*

Substances

  • Ion Channels
  • Mitochondrial Membrane Transport Proteins
  • Proto-Oncogene Proteins c-bcl-2
  • bcl-2-Associated X Protein
  • mitochondrial permeability transition pore