Transcriptional regulation of aquaporin 3 by insulin

J Cell Biochem. 2007 Nov 1;102(4):1051-8. doi: 10.1002/jcb.21350.

Abstract

In the current study, we identified a regulatory factor for the transcription of aquaporin 3 (AQP3) whose expression is repressed by insulin. We constructed a luciferase reporter vector containing bp -1382 to -12 of the 5'-flanking region of the AQP3 gene for a reporter gene assay and observed that luciferase activity in transfectants with the plasmid decreased on treatment with insulin. Serial deletion constructs revealed two regions responsible for the insulin-mediated repression, one between bps -1382 and -780, and the other between bps -404 and -82. mRNA expression of forkhead box a2 (Foxa2), the binding site of which was located between bps -1382 and -780, was found to decrease on treatment with insulin. A mutant reporter plasmid with an altered Foxa2-binding site and siRNA for the Foxa2 sequence counteracted the insulin-mediated repression of AQP3 transcription. These results suggest that Foxa2 is one of the transcriptional regulators for AQP3 gene expression regulated by insulin.

MeSH terms

  • Aquaporin 3 / genetics*
  • Base Sequence
  • Binding Sites
  • Caco-2 Cells
  • Gene Expression Regulation*
  • Genes, Reporter
  • Hepatocyte Nuclear Factor 3-beta / physiology*
  • Humans
  • Insulin / physiology*
  • Repressor Proteins
  • Transcription, Genetic*
  • Transfection

Substances

  • AQP3 protein, human
  • FOXA2 protein, human
  • Insulin
  • Repressor Proteins
  • Hepatocyte Nuclear Factor 3-beta
  • Aquaporin 3