Atrial fibrillation after aortic arch repair requiring deep hypothermic circulatory arrest: incidence, clinical outcome, and clinical predictors

J Cardiothorac Vasc Anesth. 2007 Jun;21(3):388-92. doi: 10.1053/j.jvca.2006.11.024. Epub 2007 Feb 20.

Abstract

Objective: To delineate the incidence, outcome impact, and clinical predictors of atrial fibrillation (AF) after adult aortic arch repair requiring deep hypothermic circulatory arrest (AAR-DHCA) AIMS: To determine the incidence of AF after AAR-DHCA, to determine whether AF after AAR-DHCA affects mortality or stay in the intensive care unit (ICU), to determine multivariate predictors for AF after AAR-DHCA, and to determine whether aprotinin protects against AF after AAR-DHCA.

Study design: Retrospective and observational.

Study setting: Single large university hospital.

Participants: All adults undergoing AAR-DHCA in 2000 and 2001.

Main results: The cohort size was 144. Antifibrinolytic exposure was 100%, aprotinin 66% and aminocaproic acid 34%. The incidence of AF was 34.0%. AF was not significantly associated with increased mortality or prolonged ICU stay. Advanced age was a multivariate risk factor for AF. Lower temperature nadir during DHCA was protective against postoperative AF. Aprotinin had no demonstrable effect on AF after AAR-DHCA.

Conclusions: AF after AAR-DHCA is common but does not independently increase mortality or ICU stay. The risk of AF after AAR-DHCA increases with age but decreases with the degree of hypothermia during DHCA. Aprotinin does not appear to affect the risk of AF after AAR-DHCA.

MeSH terms

  • Adult
  • Age Factors
  • Aged
  • Aorta, Thoracic / surgery*
  • Atrial Fibrillation / etiology*
  • Atrial Fibrillation / prevention & control
  • Cardiopulmonary Bypass
  • Female
  • Heart Arrest, Induced*
  • Humans
  • Incidence
  • Male
  • Middle Aged
  • Multivariate Analysis
  • Postoperative Complications / etiology*
  • Retrospective Studies
  • Risk Factors