Abstract
The aim of this investigation was to evaluate the chemopreventative/antiproliferative potential of a grape seed proanthocyanidin extract (GSPE) against colon cancer cells (CaCo2 cells) and to investigate its mechanism of action. GSPE (10-100 microg/ml) significantly inhibited cell viability and increased apoptosis in CaCo2 cells, but did not alter viability in the normal colon cell line (NCM460). The increased apoptosis observed in GSPE-treated CaCo2 cells correlated with an attenuation of PI3-kinase (p110 and p85 subunits) and decreased PKB Ser(473) phosphorylation. GSPE might thus exert its beneficial effects by means of increased apoptosis and suppression of the important PI3-kinase survival-related pathway.
Publication types
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Research Support, Non-U.S. Gov't
MeSH terms
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3-Phosphoinositide-Dependent Protein Kinases
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Antioxidants / pharmacology*
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Apoptosis / drug effects*
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Caco-2 Cells / drug effects
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Caco-2 Cells / enzymology
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Caco-2 Cells / pathology
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Cell Survival / drug effects
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Humans
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Phosphatidylinositol 3-Kinases / metabolism
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Phosphoinositide-3 Kinase Inhibitors*
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Proanthocyanidins / pharmacology*
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Protein Serine-Threonine Kinases / antagonists & inhibitors
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Protein Serine-Threonine Kinases / metabolism*
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Seeds / chemistry*
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Vitis / chemistry*
Substances
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Antioxidants
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Phosphoinositide-3 Kinase Inhibitors
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Proanthocyanidins
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proanthocyanidin
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3-Phosphoinositide-Dependent Protein Kinases
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Protein Serine-Threonine Kinases