Monovalent currents through the T-type Cav3.1 channels and their block by Mg2+

Gen Physiol Biophys. 2007 Sep;26(3):234-9.

Abstract

We have investigated the permeability of the Cav3.1 channel for Ca2+ and different monovalent cations and the block of the currents by Mg2+ ions. In the absence of extracellular divalent cations, the Cav3.1 channel was more permeable for Na+ than for Cs+ and impermeable for NMDG+. Monovalent currents were inhibited by Mg2+ of near physiological concentration by three orders of magnitude more effectively than the Ca2+ current. Inhibition of outward, but not inward current by Mg2+ was voltage-dependent. Furthermore, magnesium slowed down channel deactivation presumably by interacting with an open channel state.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Calcium / metabolism*
  • Calcium Channels, T-Type / drug effects
  • Calcium Channels, T-Type / physiology*
  • Cell Line
  • Dose-Response Relationship, Drug
  • Humans
  • Ion Channel Gating / drug effects
  • Ion Channel Gating / physiology*
  • Kidney / drug effects
  • Kidney / physiology*
  • Magnesium / administration & dosage*
  • Membrane Potentials / drug effects
  • Membrane Potentials / physiology*

Substances

  • CACNA1G protein, human
  • Calcium Channels, T-Type
  • Magnesium
  • Calcium