A novel TSHR gene mutation (Ile691Phe) in a Chinese family causing autosomal dominant non-autoimmune hyperthyroidism

J Hum Genet. 2008;53(5):475-478. doi: 10.1007/s10038-008-0257-3. Epub 2008 Feb 29.

Abstract

Familial non-autoimmune hyperthyroidism is a rare autosomal dominant genetic disease resulting from activating mutations in the thyroid-stimulating hormone receptor (TSHR) gene. In this work a Chinese family with autosomal dominant non-autoimmune hyperthyroidism across four generations was collected. The strongest evidence for linkage in this study occurred on chromosome 14q24.2-31.3. By mutation scan of the TSHR gene located within the region of interest, a heterozygote substitution (A > T) at position 2071 of the TSHR was found, changing isoleucine 691 to phenylalanine. Our study identified the first germline mutation in the intracellular C-terminal domain of TSHR.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Amino Acid Substitution / genetics*
  • Child
  • Child, Preschool
  • China
  • Female
  • Genes, Dominant*
  • Germ-Line Mutation
  • Graves Disease
  • Humans
  • Hyperthyroidism / genetics*
  • Intracellular Fluid / metabolism
  • Male
  • Mutation, Missense*
  • Pedigree
  • Protein Structure, Tertiary / genetics
  • Receptors, Thyrotropin / genetics*

Substances

  • Receptors, Thyrotropin