Abstract
Protein S (PS) is an extensively studied protein with an important function in the down-regulation of thrombin generation. Because of the presence of a pseudogene and two different forms of PS in plasma, a bound and a free form, it is one of the most difficult thrombophilias to study. A deficiency of PS predisposes subjects to (recurrent) venous thromboembolism (VTE) and foetal loss. However, the conundrum of diagnosing PS deficiency has led to conflicting reports of PS as a risk factor for VTE. In this review, we aim to present a clinical perspective of PS deficiency.
MeSH terms
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Adult
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Age of Onset
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Anticoagulants / administration & dosage
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Blood Coagulation Tests
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Blood Proteins / genetics*
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Contraceptives, Oral, Hormonal
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Contraindications
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Estrogen Replacement Therapy
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False Positive Reactions
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Female
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Heterozygote
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Humans
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Immunoassay
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Infant, Newborn
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Male
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Middle Aged
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Mutation
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Polymerase Chain Reaction / methods
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Pregnancy
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Pregnancy Complications, Hematologic / genetics*
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Pregnancy Trimesters
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Protein S / genetics
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Protein S / metabolism*
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Protein S Deficiency / diagnosis
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Protein S Deficiency / genetics*
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Protein S Deficiency / physiopathology
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Recurrence
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Risk Factors
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Thrombophilia / genetics
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Venous Thromboembolism / drug therapy
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Venous Thromboembolism / etiology*
Substances
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Anticoagulants
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Blood Proteins
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Contraceptives, Oral, Hormonal
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PROS1 protein, human
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Protein S