[Ankylosing Spondylitis and convenient nutrition]

Wien Med Wochenschr. 2008;158(9-10):294-7. doi: 10.1007/s10354-008-0536-9.
[Article in German]

Abstract

The genetic setting and immunopathogenesis of patients with ankylosing spondylitis can not be changed by the diet. Therefore, an effective pharmacological therapy always is required. As adjuvant therapy the anti-inflammatory diet can help to reduce inflammatory processes, and to delay flares of the disease. The anti-inflammatory diet is consistent with a balanced diet and prevents infirmity provoked by malnutrition. Ankylosing spondylitis is an autoimmune disease and, as such, nutrients may mimic an antigen and cause flares of the disease and increase disease activity driven by antigen reaction. This nutrient-receptor interaction occurs in the individual patient, necessitating an individual dietary counselling for the patient with ankylosing spondylitis.

Publication types

  • English Abstract

MeSH terms

  • Antioxidants / administration & dosage
  • Antioxidants / adverse effects
  • Arachidonic Acid / antagonists & inhibitors
  • Arachidonic Acid / metabolism
  • Autoimmune Diseases / diet therapy
  • Autoimmune Diseases / immunology
  • Cross Reactions
  • Eicosanoids / antagonists & inhibitors
  • Eicosanoids / biosynthesis
  • Eicosapentaenoic Acid / metabolism
  • Fatty Acids, Omega-3 / administration & dosage
  • Food Hypersensitivity / complications
  • Food Hypersensitivity / diet therapy
  • Food Hypersensitivity / immunology
  • Glutathione Peroxidase / metabolism
  • Humans
  • Nutrition Assessment
  • Selenium / administration & dosage
  • Spondylitis, Ankylosing / diet therapy*
  • Spondylitis, Ankylosing / immunology
  • Vitamin E / administration & dosage
  • Vitamin E / adverse effects
  • gamma-Linolenic Acid / administration & dosage

Substances

  • Antioxidants
  • Eicosanoids
  • Fatty Acids, Omega-3
  • Vitamin E
  • Arachidonic Acid
  • gamma-Linolenic Acid
  • Eicosapentaenoic Acid
  • Glutathione Peroxidase
  • Selenium