Axonal transport deficit in the optic nerve of rats with inherited retinitis pigmentosa and experimentally induced glaucoma

Graefes Arch Clin Exp Ophthalmol. 2008 Nov;246(11):1553-8. doi: 10.1007/s00417-008-0903-7. Epub 2008 Aug 6.

Abstract

Background: Aim of this study was to investigate the retrograde axonal transport from optic nerve (ON) to retinal ganglion cell (RGC) in two animal models: in Royal College of Surgeons (RCS) rats, a rat model for retinal degeneration, and in a rat model for glaucoma induced by elevated intraocular pressure (IOP).

Methods: To carry out this study, dextran tetramethylrhodamine (DTMR--an hydrophilic neurotracer dye) was injected into the ON; 24 hrs later, the retina was removed and the number of labeled RGCs of the experimental rats was counted and compared.

Results: The results of these studies showed that the number of fluorescent-labeled RGCs in RCS rats and in rats with elevated IOP was reduced compared to the number of labeled RGCs of their respective controls.

Conclusion: Our findings suggest that RCS rats are characterized not only by loss of photoreceptor cells but also by functional deficits of RGCs.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Axonal Transport*
  • Biological Transport, Active
  • Cell Count
  • Coloring Agents
  • Dextrans
  • Disease Models, Animal
  • Glaucoma / chemically induced
  • Glaucoma / metabolism*
  • Glaucoma / physiopathology
  • Intraocular Pressure
  • Male
  • Optic Nerve / physiopathology*
  • Rats
  • Rats, Inbred Strains
  • Rats, Sprague-Dawley
  • Retina / pathology
  • Retinal Ganglion Cells / pathology
  • Retinitis Pigmentosa / genetics
  • Retinitis Pigmentosa / metabolism*
  • Retinitis Pigmentosa / pathology
  • Rhodamines
  • Saline Solution, Hypertonic

Substances

  • Coloring Agents
  • Dextrans
  • Rhodamines
  • Saline Solution, Hypertonic
  • dextran tetramethylrhodamine